TMEM70 antibody

Cat.#: 116195

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Product Information

  • Product Name
    TMEM70 antibody
  • Documents
  • Description
    TMEM70 Rabbit Polyclonal antibody. Positive IHC detected in human heart tissue. Positive WB detected in A2780 cells. Observed molecular weight by Western-blot: 18 kDa
  • Tested applications
    ELISA, WB, IHC
  • Species reactivity
    Human; other species not tested.
  • Alternative names
    FLJ20533 antibody; TMEM70 antibody; transmembrane protein 70 antibody
  • Isotype
    Rabbit IgG
  • Preparation
    This antibody was obtained by immunization of TMEM70 recombinant protein (Accession Number: NM_017866). Purification method: Antigen affinity purified.
  • Clonality
    Polyclonal
  • Formulation
    PBS with 0.02% sodium azide and 50% glycerol pH 7.3.
  • Storage instructions
    Store at -20℃. DO NOT ALIQUOT
  • Applications

    Recommended Dilution:

    WB: 1:1000-1:10000

    IHC: 1:20-1:200

  • Validations

    A2780 cells were subjected to SDS PAGE followed by western blot with Catalog No:116195(TMEM70 antibody) at dilution of 1:800

    A2780 cells were subjected to SDS PAGE followed by western blot with Catalog No:116195(TMEM70 antibody) at dilution of 1:800

    Immunohistochemical of paraffin-embedded human heart using Catalog No:116195(TMEM70 antibody) at dilution of 1:100 (under 10x lens)

    Immunohistochemical of paraffin-embedded human heart using Catalog No:116195(TMEM70 antibody) at dilution of 1:100 (under 10x lens)

    Immunohistochemical of paraffin-embedded human heart using Catalog No:116195(TMEM70 antibody) at dilution of 1:100 (under 40x lens)

    Immunohistochemical of paraffin-embedded human heart using Catalog No:116195(TMEM70 antibody) at dilution of 1:100 (under 40x lens)

  • Background
    TMEM70 belongs to the TMEM70 family. It is involved in biogenesis of mitochondrial ATP synthase. Defects in TMEM70 are a cause of mitochondrial encephalocardiomyopathy neonatal due to ATP synthase deficiency (MT-ATPSD). A publication(PMID:21147908) identifies that TMEM70 gene defect as a pan-ethnic disorder and further redefines it as the most common cause of nuclear-origin ATP synthase deficiency.
  • References
    • Torraco A, Verrigni D, Rizza T. TMEM70: a mutational hot spot in nuclear ATP synthase deficiency with a pivotal role in complex V biogenesis. Neurogenetics. 13(4):375-86. 2012.
    • Kratochvílová H, Hejzlarová K, Vrbacký M. Mitochondrial membrane assembly of TMEM70 protein. Mitochondrion. 15:1-9. 2014.
    • Diodato D, Invernizzi F, Lamantea E. Common and Novel TMEM70 Mutations in a Cohort of Italian Patients with Mitochondrial Encephalocardiomyopathy. JIMD reports. 15:71-8. 2015.

Please note: All products are "FOR RESEARCH USE ONLY AND ARE NOT INTENDED FOR DIAGNOSTIC OR THERAPEUTIC USE"